| |
GOLF XONE™
PROFESSIONAL
GOLF DRINK
SPORTS-SPECIFIC
GOLF DRINK
THE
FIRST SPORTS DRINK DESIGNED TO
MEET THE BIOCHEMICAL NEEDS OF
THE GOLF PROFESSIONAL
|
Backed
by a United States Patent • Endorsed by Golf Legend
Billy Casper |
Designed
& Patented by World Renowned Medical Researcher:
Dr. Ann de Wees Allen® |
GOLF
XONE™
The first
and only golf drink that can make the following statements:
| • |
GLYCEMICALLY
BALANCED ENERGY |
| • |
DOWN-REGULATES
CORTISOL |
| • |
PATENTED
BUFFERED CAFFEINE |
| • |
MEETS
ALL ANTI-DOPING REGULATIONS |
| • |
UNITED
STATES PATENT |
| • |
NON-KETOGENIC |
| • |
NON-CEPHALIC |
| |
Human
Sports Performance™ |
•
HSP Approves
Golf Xone™
Click
Here
Golf Xone™ Qualifies as Safe
& Legal for Professional & Competitive
Athletes |
|
| • |
HSP Pledges One Million Dollars for Research
on Cortisol Science and Athletses Click
Here
|
| • |
Golf
Xone™ Meets Anti-Doping Guidelines for Legal
Use by Athletes |
| • |
Athletes
Against Drugs in Sports |
GOLF
XONE™
PROFESSIONAL
GOLF DRINK
SPORTS-SPECIFIC
TECHNOLOGY
|
GOLF XONE™ is
the first sports drink designed to meet the specific biochemical
requirements of the golfer. Designed
for the professional golfer, GOLF XONE™ is
the only PATENTED golf sports drink.
Designed
by Glycemic Researchers, GOLF XONE™ is the
only Certified Low Glycemic, Non-Cephalic sports
drink in the world.
INNOVATION
IN SPORTS DRINKS
The
Patented technology in GOLF XONE™ increases
absorption of the sports-specific carbohydrates by 25 percent,
which vastly improves sports performance.
GOLF
SCIENCE
Golfers walking a normal 18-hole golf course reach different
intensities of exercise, depending on their age and fitness
levels. To quantify sports performance in the golfer,
maximal oxygen uptake, maximal heart rate, and mean blood
glucose levels can be measured and quantified. This data is
utilized to create a Sports-Specific-Golf-Beverage
that meets the biochemical needs of the golfer, whether amateur
or professional.
GOLF
XONE™
SCIENCE & RESEARCH
|
GOLF-SPECIFIC-ELECTROLYTES
Golfers
of any age require Potassium (K) on a daily basis. GOLF
XONE™ contains the correct dose and form of Potassium
required for golfing activities. Sodium is contraindicated
in a gold drink, and so is any form of protein.
Calcium is not advantageous in a golf drink, as calcium metabolism
in humans requires co-ingestion with mineral-containing foods
in a meal. Further, calcium is not depleted during golf, while
Potassium is.
Though
bananas do contain Potassium, they are completely contraindicated
for golfers, as they are very high glycemic and fat-storing.
High glycemic foods or beverages reduce sports performance
and impair hand-to-eye coordination.
Sodium (salt) should only be included in fluids consumed during
aerobic or intense exercise lasting longer than 2 hours or
by individuals during any event that stimulates heavy sodium
loss (more than 3-4 g of sodium).
Playing golf does not require sodium (salt) replenishment,
and in fact, is contraindicated. That is why GOLF
XONE™ does not contain salt. Americans already consume
too much sodium/salt which causes edema.
NON-KETOGENIC
CORTISOL
& SPORTS PERFORMANCE
GOLF
XONE™ is anti-ketogenic, meaning it does
not cause ketosis. Ketogenic drinks and ketosis are
one of the primary causal factors in impaired and reduced
sports performance and hitting-the-wall syndrome
in athletes.
After
only 3 days on a high protein or ketogenic diet, high plasma
levels of Cortisol are evidenced. This negative physical reaction
to the Atkins Diet caused a plethora of researchers
and physicians to call upon the Food & Drug Administration
(FDA) to take action against the Atkins Diet.
In
response to the barrage of criticism against the Atkins
Diet, and other high protein diets, the U.S. Food
& Drug Administration published the following statement
at their official website:
“High-protein
diets, and other so-called "fad" diets are fundamentally
different from federal nutrition dietary guidelines and are
not recommended for losing weight. High-protein diets
force the kidneys to try to get rid of the excess waste products
of protein and fat, called ketones.
A buildup
of ketones in the blood (called ketosis) can cause the body
to produce high levels of uric acid, which is a risk factor
for gout (a painful swelling of the joints) and kidney stones.
Ketosis
can be especially risky for people with diabetes because it
can speed the progression of diabetic renal disease.
Ketosis
and ketogenic drinks over-elevate Cortisol, which directly
reduces performance in sports. Cortisol is a corticosteroid
hormone that has been proven to reduce sports performance,
and represents a major area of research in sports science.
Over-expression
and over-elevation of Cortisol in humans is triggered by a
variety of external biochemical events resulting in mild-to-serious
medical problems.
Stress
and ketosis are major triggers in over-elevation and stimulation
of Cortisol. Elevation of Cortisol is a major issue in athletes,
whose stress levels are considerably higher than the average
person, due to physical stress and competitive performance-driven-stress.
Athletes,
in particular, over-produce Cortisol in response
to training, competition, emotional and physical stress, peer-pressure,
and from consuming the wrong forms of protein.
One
major concern for athletes of all ages and levels, is stimulation
of Cortisol triggered by ingesting protein bars, drinks,
and supplements.
Protein
supplements over-elevate Cortisol because they instigate ketogenesis.
This
physiological reaction is controlled by the hypothalamic-pituitary-adrenal
(HPA) axis, and results in negative skeletal muscle feedback
and muscle wasting.
Ingesting Cortisol-stimulating foods, beverages and protein
drinks can cause:
| • |
Decline
and suppression of immune function |
| • |
Infertility |
| • |
Increased blood
pressure |
| • |
Increased blood
sugar levels |
| • |
Negative skeletal
muscle feedback |
Prolonged Cortisol secretion results in:
| • |
Muscle wastage |
| • |
Weight gain |
| • |
Hypoglycemia |
| • |
Inflammatory responses |
| • |
Immune suppression |
| • |
Damage to cells
in the hippocampus |
| • |
Impaired learning |
Due to the significant negative impact that
Cortisol exerts on athletes, Human Sports Performance™ (HSP)
has provided research grants for 2009-2010 focusing on the
relationship between Cortisol and sports performance.
To
that end, HSP has dedicated over $1 million dollars in 2009
to fund research in the field of Human Maximum Sports Performance,
including breakthrough research on the effects of Cortisol
on the elite athlete.
DOWNREGULATING
CORTISOL
The
February 5, 2009 Press Release issued by Encode Researchers
(www.EncodeResearch.com)
announced another groundbreaking discovery from the Encode
Dream Team, a group of world renowned physicians and
researchers:
PRESS
RELEASE
FEBRUARY 5, 2009
SCIENTISTS DISCOVER KEY TO CORTISOL DOWNREGULATION
The
scientists that pioneered the Glycemic Index
and broke the key code to the genetic disease, Sickle
Cell, announced today that they have discovered
the first pathway in humans which downregulates Cortisol.
Encode
Researchers (www.EncodeResearch.com)
have spent the past 25 years studying the hypothalamic-pituitary
axis and its relationship to aging, genetic diseases,
and L-Arginine. They received the first Glycemic patent
ever awarded, and the first patent for L-Arginine crossing
the Blood-Brain Barrier.
Their
groundbreaking research led to the discovery of Blind
Amino Acids, and the first safe transport system
for amino acids to cross the BBB, instigating the release
of the anti-aging hormone, Growth Hormone from the hypothalamus/pituitary.
It was during this research they noted that the neurohormone,
Cortisol was under the control of centers sensing blood
glucose (glycemic) levels, and was also controlled by
the hypothalamic-pituitary axis.
This
finding led to the scientists to develop a method for
de-coding Cortisol and its negative actions
on the human body. In effect, they discovered a mechanism
for mitigating the actions of Cortisol, which is known
in the biochemical arena as downregulation.
|
Following the 2009 Press
Release, the Encode Research Team began licensing
the mechanism for Cortisol Downregulation to Nutraceutical
and Pharmaceutical companies.
The
compound that allows for the downregulation of Cortisol can
be added to sports drinks, protein drinks, meal replacements,
and other products directed at athletic performance.
NO
TAURINE
In 2008, the AMERICAN HEART ASSOCIATION (AHA) announced that
energy drinks and beverages can dangerously affect heart function
and blood pressure. This announcement was made at the American
Heart Association’s Scientific Sessions and came as quite
a surprise to researchers.
The AHA report was based on clinical studies that demonstrated
“Blood pressure and heart rate levels increased in healthy
adults who drank two cans a day of a popular energy drink
containing Taurine and caffeine.”
To
view the complete Skinny Science® Newsletter
TAURINE REPORT
Click Here
Ingredient in Energy Drinks
Deemed Dangerous
|
SPORTS-SPECIFIC CARBOHYDRATES
GOLF
XONE™ contains proprietary golf-specific Low Glycemic
carbohydrates derived from fruit (SIF). These sports-specific
carbohydrates are slowly absorbed in the gut, providing
a "time-released" matrix which delivers
energy at a gradual rate.
Carbohydrates
are different forms of sugars linked together in polymers,
which break down into glucose, the primary fuel of the body.
All fruits and vegetables are carbohydrates.
Sports
drinks that do not contain appropriate amounts of carbohydrate
promote sports-failure due to glycogen
depletion and hypoglycemia.
BRAIN REQUIREMENTS
The human brain requires a high amount of carbohydrates to
function, and uses about two-thirds of all carbohydrates found
in the bloodstream (serum levels) to maintain normal functions.
Sports drinks that do not contain adequate carbohydrates lead
to reduction in the brain’s ability to provide cognitive
function mandatory in sports events, and reduce hand-to-eye
coordination, as well as focus.
ACCESSING GOLF-SPORTS FUEL SOURCES
According to the Journal of Sports Science “Athletes
will benefit the most by tailoring their individual needs
for water, carbohydrate and salt to the specific challenges
of their sport, especially considering the environment's impact
on sweating and heat stress.”
In designing an appropriate and functional golf drink, it
is imperative to tailor the drink to match the individual
needs of the golfer. This requires an in-depth understanding
of human biochemistry and fuel-sources related to the game
of golf.
Golfers do not deplete muscle glycogen during
golfing activities, so to give a golfer a drink that is designed
for sports that actually does deplete muscle glycogen (such
as long-distance runners), creates reduced sports performance,
as well as increased belly fat.
Golf is categorized as a Class I sport by
Human Maximum Performance™, in terms of biochemical
mechanisms, and as such, warrants a specific carbohydrate
structure and glycemic response.
Glycogen is the “emergency fuel” stored in the muscles and
liver from glucose. It is considered “emergency fuel” because
the body’s ability to store carbohydrates is extremely limited
and is used during energy shortages.
Only 300-400 grams of carbohydrate can be stored in muscles
and 60-90 grams can be stored in the liver for glucose conversion.
This equates to about one serving of spaghetti (2 cups cooked),
which is a very conservative supply of energy.
When the muscles and liver are glutted with glycogen, excess
carbohydrates are then converted to fat and stored in adipose
tissue fat cells. High Glycemic carbohydrates are shunted
into fat cells via Lipoprotein Lipase (LPL), the Gatekeeper
for Fat-Storage in the Fat Cell, while Low Glycemic carbohydrates
are utilized and burned as energy.
As the brain uses and requires fuel, liver glycogen is broken
down and sent to the brain. Since the body’s ability to store
carbohydrates is limited, a constant supply is required to
function normally.
GOLF
XONE™ is specifically tailored to the
biochemical needs of the golfer. |
GOLF
XONE™
PROFESSIONAL GOLF DRINK
TOOLS
FOR WEIGHT MANAGEMENT
|
It
is estimated that in 2009 (Golf Digest) 66 percent
of golfers are overweight. In 2008, it was calculated that
46 percent of golfers were overweight.
The
rise in obesity and weight gain in golfers of 46 % in 2008
to 66 % in 2009 is cause for concern.
"The results almost made us drop our hot dogs and
hi-cal drinks and sit up in our Barcaloungers:
Quite simply, we're in terrible shape," lamented
Golf Digest, reporting on their survey of 514 golfers
(80% male, average age 52.7).
Among respondents, 19% have never set foot in a gym; 18% always
use a cart; 29% buy hot dogs and 16% buy a beer on the course,
while 9% have drunk 10 or more beers during a round.
Data published at the PGATOUR Official Website (www.PGATOUR.com)
states that the National Golf Foundation had calculated
2008 statistics on golfers and weight:
• 46 percent
of golfers
are overweight
and 27 percent are obese |
Therefore, GOLF XONE™ researchers
elected to help combat the increasing levels of body fat in
golfers by engineering-in specific fat-burning components
to the golf drink.
Golfers are not the only group of American’s whose body fat
levels are increasing, as their spouses are statistically
likely to be overweight. To that end, GOLF XONE™
may also be used by the rest of the adult family as an adjunct
to weight management, as long as they are over age 16.
GOLF XONE™
Mechanisms related to addressing excess body fat in
humans
The biochemical mechanisms which help address excess body
fat in the GOLF XONE™ drink are:
| 1)
|
Thermogenic Fat-Burning |
| 2) |
Down-Regulated
Lipoprotein Lipase (LPL) |
| 3) |
Down-Regulated
Cortisol |
| 4) |
Controlled Glycemic
and Insulin Parameters |
THERMOGENIC FAT BURNING
Single-dose oral administration of 1 serving of the Buffered
Caffeine Patented Compound (TPC) (found in one serving of
GOLF XONE™) increased resting metabolic rate
of both lean and post-obese human subjects and improved defective
diet-induced thermogenesis (DIT); significant influence on
energy balance; promotes additional thermogenic fat-burning
in the treatment of the obese.
| • |
Increases energy
expenditure by 8-11% |
| • |
Increases Thermogenic Fat-Burning up to 500% in dose
dependent fashion |
DownRegulation of Lipoprotein Lipase
(LPL)
Adipose Tissue Fat and Lipoprotein Lipase (LPL) are directly
related, with LPL activation playing a major role in the accumulation
of adipose tissue fat. Higher LPL activity and/or LPL stimulation
causes increases in body fat, particularly in women.
Visceral adipocytes are highly sensitive to insulin and are
stimulated by high blood glucose and insulin levels in humans.
Engorged visceral fat stores flood portal circulation with
free fatty acids at metabolically inappropriate times when
fatty acids are unlikely to be oxidized, thus exposing tissues
to excessive free fatty acid levels and giving rise to insulin
resistance syndrome.
Down-regulating LPL activity can largely prevent and/or correct
visceral obesity. A sound efficacious biochemical approach
to excess adipose tissue fat is down-regulating LPL activity
in visceral adipocytes.
The ability of alpha-glucosidase inhibitor therapy to selectively
reduce visceral adiposity is evidence that down-regulation
of diurnal insulin secretion has a greater impact on LPL activity
in visceral fat than in subcutaneous fat.
Low Glycemic beverages, compounds, and diets decrease visceral
LPL activity. Pre-administration of a Low Glycemic, thermogenic
compound placed in coffee or tea, or any other ingested oral
delivery system, suppresses transcription of LPL, resulting
in a favorable impact in reducing the proclivity of visceral
obesity in humans.
The relationship between insulin resistance and obesity is
well established, and an increase in obesity is directly implicated
in increased incidence of diabetes. Visceral adiposity has
been particularly associated with insulin resistance. Insulin
resistance of the liver derives from a relative increase in
the delivery of free fatty acids (FFA) from the omental fat
depot to the liver (via the portal vein). Increased delivery
results from
| 1) |
more stored lipids
in omental depot |
| 2) |
severe insulin
resistance of the central fat depot |
| 3) |
regulation of
visceral lipolysis by the central nervous system |
Insulin regulates liver glucose output primarily via control
of adipocyte lipolysis, rendering the liver relatively
insulin resistant.
Enhanced B-cell sensitivity to glucose is a compensation mechanism,
along with reduced liver insulin clearance, which allows for
a greater fraction of B-cell insulin secretion to bypass liver
degradation, resulting in hyper-insulinemic compensation.
The
Journal of Clinical Endocrinology Metabolism, 2002 Sep;87(9):4166-70;
Differences in adipose tissue metabolism between postmenopausal
and perimenopausal women, illustrated the strong capacity
of LPL to induce body fat gains and weight gain. Researchers
compared abdominal and gluteal adipose tissue metabolism [basal
and stimulated lipolysis and activity of adipose tissue lipoprotein
lipase (AT-LPL)] in post-menopausal and peri-menopausal women.
Women in menopause experience reduced fat-burning due to hormonal
changes. The researchers found that LPL activity is significantly
higher in post-menopausal women compared with peri-menopausal
women in both gluteal (4.9 +/- 3.6 vs. 2.0 +/- 1.4 nmol free
fatty acid/g.min; P < 0.05) and abdominal (3.2 +/- 2.6
vs. 1.3 +/- 0.9 nmol free fatty acid/g.min; P < 0.05) adipose
cells. Researchers stated that “The lower lipolysis (fat-burning)
and higher LPL activity in post-menopausal women predisposes
them to gain body fat after menopause.”
LPL
IS STIMULATED BY INSULIN
The
2008 Journal of Lipid Research clearly showed that
LPL is “Regulated by insulin.” Further, that LPL is also regulated
by fasting, which explains why fasting (consuming low calories)
increases adipose tissue fat cells and coordinates bio-distribution
of fat-derived calories.
J
Lipid Res. 2008 Nov 24, Regulation of fatty acid uptake into
tissues: lipoprotein lipase- and CD36-mediated pathways
LPL
The efficacy of GOLF XONE™ is enhanced by
its ability to down-regulate Lipoprotein Lipase (LPL). Currently,
GOLF XONE™ is the only golf drink
on the market that down-regulates LPL and CORTISOL.
GOLF XONE™ ENERGY MATRIX
GOLF XONE™ contains a specially formulated
Energy Matrix that provides sustained and balanced high energy
levels. GOLF XONE™ contains a unique energetic
blend designed to provide maximum energy enhancement in humans
including:
| • |
Patented Buffered
Caffeine |
| • |
High-Energy Matrix |
| • |
Certified Balanced-Energy
Low Glycemic Fruit Base |
| • |
Green Tea with
ECGC |
| • |
Niacin-Bound
Chromium |
PRODUCTION OF HIGHER ENERGY LEVELS:
THE MECHANISM
GOLF
XONE™ delivers high energy levels and alertness in
humans, not by speeding you up, but by keeping you
from slowing down.
Each
time brain cells fire, they product a squirt of a
chemical that serves as an off-switch that keeps neural activity
in check.
GOLF
XONE™ biochemically blocks the chemical, and jams
the switch, so it can’t be turned down. This energy-effect
can last from 2-4 hours.
Additionally,
the time-released fruit glycoside system unique to the formula
remains in the intestinal tract for a long period, providing
increased energy over a 2-4 hour duration.
5-HOUR
ENERGY CLAIMS: HIGH TECH IGNORANCE
Energy
products claiming to deliver energy for more than a 2-4 hour
period are absurd. The human brain and body function on a
4-hour time clock in terms of energy capacity, and no
product can sustain more than 4-hours of energy. It is
physically impossible.
5-hour
energy claims are an example of High Tech Ignorance.
GOLF XONE™ scientists refuse to make false
and misleading claims and demand accurate scientific validation.
PHYSIOLOGICAL
EFFECTS: FULL DISCLOSURE SCIENCE
GOLF
XONE™ is effective at increasing energy, enacting
thermogenesis via DIT, and adipose-tissue-fat-burning (ATFB),
via the following mechanisms:
| •
|
Stimulates
under-functioning adrenal glands by increasing the output
of epinephrine and norepinephrine from the adrenal glands.
|
| •
|
Stimulates
the release of catecholamines from the adrenal medulla
and releases catecholamines due to a central action
by and affecting C-AMP. |
| •
|
Donates
methyl groups in anabolic processes (Rivici Index).
|
| •
|
Increases
energy expenditure dose dependently. Stepwise regression
analysis with the thermogenic response as the dependent
variable yields the following equation: (proprietary)
as provided to our FDA-Regulatory attorneys for
GOLF XONE™ Thermogenics validation
of efficacy. |
| •
|
Turns
on Brown-Fat-Thermogenesis in humans. |
| •
|
Removes
adenos and phosphodiesterase blocks to thermogenesis
in humans. |
| •
|
Triggers
noradrenalin to active thermogenesis in isolated brown
adipocytes by stimulating lipolysis. |
| •
|
Inclusion
of Low Glycemic, thermogenic carbohydrates and PP glycosides
that blunt Lipoprotein Lipase (LPL). |
| •
|
Inclusion
of Buffered Caffeine (full U.S. Patent) that does not
exhibit the adipose-tissue fat-storing properties of
regular caffeine. |
BODY
OF RESEARCH
Human In Vivo Clinical Research & Trials
| • |
Anti-Carbohydrate properties, clinical studies, and
metabolic effects in humans. |
| • |
Low
Glycemic Responses to specific Anti-Carbohydrate Fruit
Sweeteners; Human In Vivo Clinical Trials. |
| • |
Blood glucose effect of sugars, sweeteners and carbohydrates,
circulating adiponectin (ACRP30), metabolic syndrome
and sweeteners, genetic mutations in the leptin gene,
adipocyte glucose metabolism, decreased glucose-induced
thermogenesis (DGIT), circulating C peptide concentrations
and insulin resistance, substrate utilization of carbohydrates
and sweeteners. |
| • |
The Insulin-Leptin-Ghrelin relationship in energy metabolism,
improvement of glucose tolerance in type 2 diabetics
in response to acute catalytic low-dose fruit glycosides,
muscle glycogen and carbohydrates, Liporotein Lipase
(LPL) and sugars. |
| • |
Fruit Glycosides that do not trigger adipose tissue
fat-storage, diabetes, or insulin resistance in humans,
pathogenesis of obesity and Diet-Induced-Thermogenic
(DIT) agents in humans. |
| • |
Thermogenic and fat-storing response of carbohydrates,
sugars and sweeteners, stimulation of fat-storing enzymes
in humans, Brown Adipose Tissue (BAT) and thermogenesis,
internal vs DIT thermogenesis, thermogenic capacity
of cells and tissues. |
| • |
UCP
in mitochondria, Resistin, lipolytic actions in humans,
appestat centers of the brain, identification and reduction
of fat cell mass in humans, caloric conversion info
fat cells, human genetic code related to deposition
of adipose tissue body fat, N.E.A.T., fat thermostat
in humans, hypothalamus-related fat-storage, cellular
level thermogenesis, caffeine and DIT. |
| • |
Methods for buffering caffeine to eliminate fat-storage
in fat cells, caffeine thermogenesis, LPL gatekeepers
for fat-storage in the fat cell, chromium and thermogenesis,
chocolate and Serotonin-response in human female population. |
| • |
Agents
that activate Serotonin, high-protein diets and reduced
thermogenesis, aging and adipose tissue fat accumulation,
adipocyte lineage, regulation of beta-3-adrenoceptor
expression in white vs brown fat cells. |
PATENTED THERMOGENIC FAT-BURNING
The
form of thermogenesis in humans activated by GOLF
XONE™ is called Diet-Induced-Thermogenesis
(DIT) and is well known in the scientific literature. GOLF
XONE™ produces a DIT thermogenic effect, de novo lipogenesis,
as well as carbohydrate oxidation (burning) in a Proprietary
Low Glycemic matrix.
ANTI-GLUCOSE MATRIX
GOLF XONE™ contains a specially formulated
ANTI-GLUCOSE MATRIX that addresses key areas in fat-burning
metabolism:
| • |
Helps produce the neurotransmitter serotonin, which
is related to positive moods. |
| • |
Provides vital serum support in the Glucose-Producing-Reaction
(GPR). |
In order to combat Stress-Related-Eating (SRE)
and False-Food-Cravings, the human body requires the opposite
of Glucose-elevation, which is a Low Glycemic Matrix.
A Low Glycemic Matrix does not stimulate glucose, and conversely
blocks False-Food-Cravings. The unique Matrix found in GOLF
XONE™ provides a mechanism for blocking fat-storage
and glucose-fat-storage.
The human body, and specifically the brain, requires at least
130 grams of carbohydrates per day for cognitive function
and energy. Low energy levels and impaired cognitive function
are typical when following a low carb diet.
High glycemic carbohydrates cause glucose and blood sugar
swings, fat-storage, and weight gain.
High protein, and low carb diets and foods, exacerbate (worsen)
the biochemical stress machine by depriving the body of stress-calming
carbohydrates.
Thermogenic drinks and sports drink and compounds that do
not contain a Low Glycemic Matrix cause fat-storage.
GOLF XONE™
PRODUCT USE GUIDELINES & CONTRAINDICATIONS
SUGGESTED
USE:
Intended
for adult use only. Not for use by individuals under 16 years
of age. Not for use by children or by pregnant or lactating
women. Use only as directed. Not for use by anyone sensitive
to any of the ingredients.
This
is a Sports-Specific beverage designed exclusively for Golfers
and/or Class I Athletes (non-aerobic exercise, such as Golf,
Race-Car-Drivers, Bodybuilders, Powerlifters). Not to be used
for aerobic or intense sports or exercise.
AS
A BEVERAGE FOR GOLFERS: Add one serving (one packet)
to 16 ounces of plain bottled water (not flavored water or
sports drinks) or to 16 ounces (oz) of plain water. Do not
use more than 1 serving at a time. Do not use more than 4
servings total per day.
WEIGHT
MANAGEMENT SUPPORT: GOLF XONE™ drink supports weight
management in multiple ways: (1) Downregulation of Cortisol
(2) Preferentially burns fat over carbohydrates (3) Downregulation
of LPL (4) Non-Cephalic. Protocol for weight management support:
Take 1 serving 30 minutes prior to a meal or in-between meals
(up to 4 servings per day).
If
dissolving into a full bottle of water, take a few sips before
adding this product.
The
energy-enhancement features of GOLF XONE™
may interfere with sleep if taken too close to bedtime. To
avoid this, do not ingest after dinner or too close to sleep-hours.
PLEASE
USE THIS PRODUCT RESPONSIBLY. Do not mix with alcohol or alcoholic
drinks.
CAUTIONS:
Do not exceed recommended serving size. Exceeding the recommended
serving has not been shown to result in greater effectiveness.
DO NOT USE IF YOU are pregnant or nursing. Keep out of reach
of children. Do not use prior to intense aerobic sports or
exercise.
CONSULT
A PHYSICIAN BEFORE USING THIS PRODUCT IF YOU: Take
a prescription medication, are sensitive to the effects of
caffeine, or are allergic to any of the ingredients in this
product.
Do
not drink coffee or use caffeine products within 2 hours of
taking this product. It is advisable to reduce consumption
of products containing caffeine while taking this product.
If you have a history of any of the following: heart disease,
thyroid disease, diabetes, high blood pressure, glaucoma,
difficulty in urinating or prostate enlargement, consult with
your physician prior to using this product. DISCONTINUE
USE AND CONSULT A PHYSICIAN IF YOU: Experience rapid
heartbeat, dizziness, severe headache, shortness of breath
or other similar symptoms.
|
(1) American Heart Journal
Volume 152, Issue 3, September 2006
Al-Habori M, Peak M, Thomas TH, and Agius L. The role of cell
swelling in the stimulation of glycogen synthesis by insulin.
Biochem J 282: 789–796, 1992
Gardner, L. and Reiser, S. "Effects of Dietary Carbohydrate
on Fasting Levels of Human Growth Hormone and Cortisol."
Proceedings of the Society for Experimental Biology and Medicine.
1982;169:36-40.
Angus DJ, Febbraio MA, and Hargreaves M. Plasma glucose kinetics
during prolonged exercise in trained humans when fed carbohydrate.
Am J Physiol Endocrinol Metab 283: E573–E577, 2002.
J Sports Sci. 2004 Jan;22(1):39-55. Fluid and fuel intake
during exercise.
Battram DS, Shearer J, Robinson D, and Graham TE. Caffeine
ingestion does not impede the resynthesis of proglycogen and
macroglycogen after prolonged exercise and carbohydrate supplementation
in humans. J Appl Physiol 96: 943–950, 2004.
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