GOLF XONE™

PROFESSIONAL GOLF DRINK

SPORTS-SPECIFIC GOLF DRINK

THE FIRST SPORTS DRINK DESIGNED TO
MEET THE BIOCHEMICAL NEEDS OF
THE GOLF PROFESSIONAL



Backed by a United States Patent • Endorsed by Golf Legend Billy Casper
Designed & Patented by World Renowned Medical Researcher:
Dr. Ann de Wees Allen®


GOLF XONE™
The first and only golf drink that can make the following statements:

GLYCEMICALLY BALANCED ENERGY
DOWN-REGULATES CORTISOL
PATENTED BUFFERED CAFFEINE
MEETS ALL ANTI-DOPING REGULATIONS
UNITED STATES PATENT
NON-KETOGENIC
NON-CEPHALIC



 
Human Sports Performance™
HSP Approves Golf Xone™
Click Here

Golf Xone™ Qualifies as Safe
& Legal for Professional & Competitive
Athletes

 
HSP Pledges One Million Dollars for Research on Cortisol Science and Athletses Click Here 
  Golf Xone™ Meets Anti-Doping Guidelines for Legal Use by Athletes
  Athletes Against Drugs in Sports



GOLF XONE™

PROFESSIONAL GOLF DRINK

SPORTS-SPECIFIC TECHNOLOGY




GOLF XONE™
is the first sports drink designed to meet the specific biochemical requirements of the golfer.

Designed for the professional golfer, GOLF XONE™ is the only PATENTED golf sports drink.

Designed by Glycemic Researchers, GOLF XONE™ is the only Certified Low Glycemic, Non-Cephalic sports drink in the world.

INNOVATION IN SPORTS DRINKS

The Patented technology in GOLF XONE™ increases absorption of the sports-specific carbohydrates by 25 percent, which vastly improves sports performance.

GOLF SCIENCE

Golfers walking a normal 18-hole golf course reach different intensities of exercise, depending on their age and fitness levels. To quantify sports performance in the golfer, maximal oxygen uptake, maximal heart rate, and mean blood glucose levels can be measured and quantified. This data is utilized to create a Sports-Specific-Golf-Beverage that meets the biochemical needs of the golfer, whether amateur or professional.





GOLF XONE™
SCIENCE & RESEARCH



GOLF-SPECIFIC-ELECTROLYTES

Golfers of any age require Potassium (K) on a daily basis. GOLF XONE™ contains the correct dose and form of Potassium required for golfing activities. Sodium is contraindicated in a gold drink, and so is any form of protein. Calcium is not advantageous in a golf drink, as calcium metabolism in humans requires co-ingestion with mineral-containing foods in a meal. Further, calcium is not depleted during golf, while Potassium is.

Though bananas do contain Potassium, they are completely contraindicated for golfers, as they are very high glycemic and fat-storing. High glycemic foods or beverages reduce sports performance and impair hand-to-eye coordination.

Sodium (salt) should only be included in fluids consumed during aerobic or intense exercise lasting longer than 2 hours or by individuals during any event that stimulates heavy sodium loss (more than 3-4 g of sodium).

Playing golf does not require sodium (salt) replenishment, and in fact, is contraindicated. That is why GOLF XONE™ does not contain salt. Americans already consume too much sodium/salt which causes edema.



NON-KETOGENIC

CORTISOL & SPORTS PERFORMANCE

GOLF XONE™ is anti-ketogenic, meaning it does not cause ketosis. Ketogenic drinks and ketosis are one of the primary causal factors in impaired and reduced sports performance and hitting-the-wall syndrome in athletes.

After only 3 days on a high protein or ketogenic diet, high plasma levels of Cortisol are evidenced. This negative physical reaction to the Atkins Diet caused a plethora of researchers and physicians to call upon the Food & Drug Administration (FDA) to take action against the Atkins Diet.

In response to the barrage of criticism against the Atkins Diet, and other high protein diets, the U.S. Food & Drug Administration published the following statement at their official website:

“High-protein diets, and other so-called "fad" diets are fundamentally different from federal nutrition dietary guidelines and are not recommended for losing weight. High-protein diets force the kidneys to try to get rid of the excess waste products of protein and fat, called ketones.

A buildup of ketones in the blood (called ketosis) can cause the body to produce high levels of uric acid, which is a risk factor for gout (a painful swelling of the joints) and kidney stones.

Ketosis can be especially risky for people with diabetes because it can speed the progression of diabetic renal disease.

Ketosis and ketogenic drinks over-elevate Cortisol, which directly reduces performance in sports. Cortisol is a corticosteroid hormone that has been proven to reduce sports performance, and represents a major area of research in sports science.

Over-expression and over-elevation of Cortisol in humans is triggered by a variety of external biochemical events resulting in mild-to-serious medical problems.

Stress and ketosis are major triggers in over-elevation and stimulation of Cortisol. Elevation of Cortisol is a major issue in athletes, whose stress levels are considerably higher than the average person, due to physical stress and competitive performance-driven-stress.

Athletes, in particular, over-produce Cortisol in response to training, competition, emotional and physical stress, peer-pressure, and from consuming the wrong forms of protein.

One major concern for athletes of all ages and levels, is stimulation of Cortisol triggered by ingesting protein bars, drinks, and supplements.

Protein supplements over-elevate Cortisol because they instigate ketogenesis.

This physiological reaction is controlled by the hypothalamic-pituitary-adrenal (HPA) axis, and results in negative skeletal muscle feedback and muscle wasting.


Ingesting Cortisol-stimulating foods, beverages and protein drinks can cause:

Decline and suppression of immune function
Infertility
Increased blood pressure
Increased blood sugar levels
Negative skeletal muscle feedback


Prolonged Cortisol secretion results in:

Muscle wastage
Weight gain
Hypoglycemia
Inflammatory responses
Immune suppression
Damage to cells in the hippocampus
Impaired learning


Due to the significant negative impact that Cortisol exerts on athletes, Human Sports Performance™ (HSP) has provided research grants for 2009-2010 focusing on the relationship between Cortisol and sports performance.

To that end, HSP has dedicated over $1 million dollars in 2009 to fund research in the field of Human Maximum Sports Performance, including breakthrough research on the effects of Cortisol on the elite athlete.


DOWNREGULATING CORTISOL

The February 5, 2009 Press Release issued by Encode Researchers (www.EncodeResearch.com) announced another groundbreaking discovery from the Encode Dream Team, a group of world renowned physicians and researchers:

PRESS RELEASE
FEBRUARY 5, 2009
SCIENTISTS DISCOVER KEY TO CORTISOL DOWNREGULATION

The scientists that pioneered the Glycemic Index and broke the key code to the genetic disease, Sickle Cell, announced today that they have discovered the first pathway in humans which downregulates Cortisol.

Encode Researchers (www.EncodeResearch.com) have spent the past 25 years studying the hypothalamic-pituitary axis and its relationship to aging, genetic diseases, and L-Arginine. They received the first Glycemic patent ever awarded, and the first patent for L-Arginine crossing the Blood-Brain Barrier.

Their groundbreaking research led to the discovery of Blind Amino Acids, and the first safe transport system for amino acids to cross the BBB, instigating the release of the anti-aging hormone, Growth Hormone from the hypothalamus/pituitary. It was during this research they noted that the neurohormone, Cortisol was under the control of centers sensing blood glucose (glycemic) levels, and was also controlled by the hypothalamic-pituitary axis.

This finding led to the scientists to develop a method for de-coding Cortisol and its negative actions on the human body. In effect, they discovered a mechanism for mitigating the actions of Cortisol, which is known in the biochemical arena as downregulation.


Following the 2009 Press Release, the Encode Research Team began licensing the mechanism for Cortisol Downregulation to Nutraceutical and Pharmaceutical companies.

The compound that allows for the downregulation of Cortisol can be added to sports drinks, protein drinks, meal replacements, and other products directed at athletic performance.


NO TAURINE

In 2008, the AMERICAN HEART ASSOCIATION (AHA) announced that energy drinks and beverages can dangerously affect heart function and blood pressure. This announcement was made at the American Heart Association’s Scientific Sessions and came as quite a surprise to researchers.

The AHA report was based on clinical studies that demonstrated “Blood pressure and heart rate levels increased in healthy adults who drank two cans a day of a popular energy drink containing Taurine and caffeine.”



To view the complete Skinny Science® Newsletter
TAURINE REPORT
Click Here

Ingredient in Energy Drinks
Deemed Dangerous


 



SPORTS-SPECIFIC CARBOHYDRATES

GOLF XONE™ contains proprietary golf-specific Low Glycemic carbohydrates derived from fruit (SIF). These sports-specific carbohydrates are slowly absorbed in the gut, providing a "time-released" matrix which delivers energy at a gradual rate.

Carbohydrates are different forms of sugars linked together in polymers, which break down into glucose, the primary fuel of the body. All fruits and vegetables are carbohydrates.

Sports drinks that do not contain appropriate amounts of carbohydrate promote sports-failure due to glycogen depletion and hypoglycemia.



BRAIN REQUIREMENTS

The human brain requires a high amount of carbohydrates to function, and uses about two-thirds of all carbohydrates found in the bloodstream (serum levels) to maintain normal functions. Sports drinks that do not contain adequate carbohydrates lead to reduction in the brain’s ability to provide cognitive function mandatory in sports events, and reduce hand-to-eye coordination, as well as focus.



ACCESSING GOLF-SPORTS FUEL SOURCES

According to the Journal of Sports Science “Athletes will benefit the most by tailoring their individual needs for water, carbohydrate and salt to the specific challenges of their sport, especially considering the environment's impact on sweating and heat stress.”

In designing an appropriate and functional golf drink, it is imperative to tailor the drink to match the individual needs of the golfer. This requires an in-depth understanding of human biochemistry and fuel-sources related to the game of golf.

Golfers do not deplete muscle glycogen during golfing activities, so to give a golfer a drink that is designed for sports that actually does deplete muscle glycogen (such as long-distance runners), creates reduced sports performance, as well as increased belly fat.

Golf is categorized as a Class I sport by Human Maximum Performance™, in terms of biochemical mechanisms, and as such, warrants a specific carbohydrate structure and glycemic response.

Glycogen is the “emergency fuel” stored in the muscles and liver from glucose. It is considered “emergency fuel” because the body’s ability to store carbohydrates is extremely limited and is used during energy shortages.

Only 300-400 grams of carbohydrate can be stored in muscles and 60-90 grams can be stored in the liver for glucose conversion. This equates to about one serving of spaghetti (2 cups cooked), which is a very conservative supply of energy.

When the muscles and liver are glutted with glycogen, excess carbohydrates are then converted to fat and stored in adipose tissue fat cells. High Glycemic carbohydrates are shunted into fat cells via Lipoprotein Lipase (LPL), the Gatekeeper for Fat-Storage in the Fat Cell, while Low Glycemic carbohydrates are utilized and burned as energy.

As the brain uses and requires fuel, liver glycogen is broken down and sent to the brain. Since the body’s ability to store carbohydrates is limited, a constant supply is required to function normally.

GOLF XONE™ is specifically tailored to the
biochemical needs of the golfer.




GOLF XONE™
PROFESSIONAL GOLF DRINK

TOOLS FOR WEIGHT MANAGEMENT



It is estimated that in 2009 (Golf Digest) 66 percent of golfers are overweight. In 2008, it was calculated that 46 percent of golfers were overweight.

The rise in obesity and weight gain in golfers of 46 % in 2008 to 66 % in 2009 is cause for concern.

"The results almost made us drop our hot dogs and hi-cal drinks and sit up in our Barcaloungers: Quite simply, we're in terrible shape," lamented Golf Digest, reporting on their survey of 514 golfers (80% male, average age 52.7).

Among respondents, 19% have never set foot in a gym; 18% always use a cart; 29% buy hot dogs and 16% buy a beer on the course, while 9% have drunk 10 or more beers during a round.

Data published at the PGATOUR Official Website (www.PGATOUR.com) states that the National Golf Foundation had calculated 2008 statistics on golfers and weight:

• 46 percent of golfers are overweight
and 27 percent are obese


Therefore, GOLF XONE™ researchers elected to help combat the increasing levels of body fat in golfers by engineering-in specific fat-burning components to the golf drink.

Golfers are not the only group of American’s whose body fat levels are increasing, as their spouses are statistically likely to be overweight. To that end, GOLF XONE™ may also be used by the rest of the adult family as an adjunct to weight management, as long as they are over age 16.

GOLF XONE™

Mechanisms related to addressing excess body fat in humans

The biochemical mechanisms which help address excess body fat in the GOLF XONE™ drink are:

1) Thermogenic Fat-Burning
2) Down-Regulated Lipoprotein Lipase (LPL)
3) Down-Regulated Cortisol
4) Controlled Glycemic and Insulin Parameters


THERMOGENIC FAT BURNING

Single-dose oral administration of 1 serving of the Buffered Caffeine Patented Compound (TPC) (found in one serving of GOLF XONE™) increased resting metabolic rate of both lean and post-obese human subjects and improved defective diet-induced thermogenesis (DIT); significant influence on energy balance; promotes additional thermogenic fat-burning in the treatment of the obese.

Increases energy expenditure by 8-11%
Increases Thermogenic Fat-Burning up to 500% in dose dependent fashion


DownRegulation of Lipoprotein Lipase (LPL)

Adipose Tissue Fat and Lipoprotein Lipase (LPL) are directly related, with LPL activation playing a major role in the accumulation of adipose tissue fat. Higher LPL activity and/or LPL stimulation causes increases in body fat, particularly in women.

Visceral adipocytes are highly sensitive to insulin and are stimulated by high blood glucose and insulin levels in humans.

Engorged visceral fat stores flood portal circulation with free fatty acids at metabolically inappropriate times when fatty acids are unlikely to be oxidized, thus exposing tissues to excessive free fatty acid levels and giving rise to insulin resistance syndrome.

Down-regulating LPL activity can largely prevent and/or correct visceral obesity. A sound efficacious biochemical approach to excess adipose tissue fat is down-regulating LPL activity in visceral adipocytes.

The ability of alpha-glucosidase inhibitor therapy to selectively reduce visceral adiposity is evidence that down-regulation of diurnal insulin secretion has a greater impact on LPL activity in visceral fat than in subcutaneous fat.

Low Glycemic beverages, compounds, and diets decrease visceral LPL activity. Pre-administration of a Low Glycemic, thermogenic compound placed in coffee or tea, or any other ingested oral delivery system, suppresses transcription of LPL, resulting in a favorable impact in reducing the proclivity of visceral obesity in humans.

The relationship between insulin resistance and obesity is well established, and an increase in obesity is directly implicated in increased incidence of diabetes. Visceral adiposity has been particularly associated with insulin resistance. Insulin resistance of the liver derives from a relative increase in the delivery of free fatty acids (FFA) from the omental fat depot to the liver (via the portal vein). Increased delivery results from

1) more stored lipids in omental depot
2) severe insulin resistance of the central fat depot
3) regulation of visceral lipolysis by the central nervous system


Insulin regulates liver glucose output primarily via control of adipocyte lipolysis, rendering the liver relatively insulin resistant.

Enhanced B-cell sensitivity to glucose is a compensation mechanism, along with reduced liver insulin clearance, which allows for a greater fraction of B-cell insulin secretion to bypass liver degradation, resulting in hyper-insulinemic compensation.

The Journal of Clinical Endocrinology Metabolism, 2002 Sep;87(9):4166-70; Differences in adipose tissue metabolism between postmenopausal and perimenopausal women, illustrated the strong capacity of LPL to induce body fat gains and weight gain. Researchers compared abdominal and gluteal adipose tissue metabolism [basal and stimulated lipolysis and activity of adipose tissue lipoprotein lipase (AT-LPL)] in post-menopausal and peri-menopausal women. Women in menopause experience reduced fat-burning due to hormonal changes. The researchers found that LPL activity is significantly higher in post-menopausal women compared with peri-menopausal women in both gluteal (4.9 +/- 3.6 vs. 2.0 +/- 1.4 nmol free fatty acid/g.min; P < 0.05) and abdominal (3.2 +/- 2.6 vs. 1.3 +/- 0.9 nmol free fatty acid/g.min; P < 0.05) adipose cells. Researchers stated that “The lower lipolysis (fat-burning) and higher LPL activity in post-menopausal women predisposes them to gain body fat after menopause.”

LPL IS STIMULATED BY INSULIN

The 2008 Journal of Lipid Research clearly showed that LPL is “Regulated by insulin.” Further, that LPL is also regulated by fasting, which explains why fasting (consuming low calories) increases adipose tissue fat cells and coordinates bio-distribution of fat-derived calories.

J Lipid Res. 2008 Nov 24, Regulation of fatty acid uptake into tissues: lipoprotein lipase- and CD36-mediated pathways

LPL

The efficacy of GOLF XONE™ is enhanced by its ability to down-regulate Lipoprotein Lipase (LPL). Currently, GOLF XONE™ is the only golf drink on the market that down-regulates LPL and CORTISOL.

GOLF XONE™ ENERGY MATRIX

GOLF XONE™ contains a specially formulated Energy Matrix that provides sustained and balanced high energy levels. GOLF XONE™ contains a unique energetic blend designed to provide maximum energy enhancement in humans including:

Patented Buffered Caffeine
High-Energy Matrix
Certified Balanced-Energy Low Glycemic Fruit Base
Green Tea with ECGC
Niacin-Bound Chromium


PRODUCTION OF HIGHER ENERGY LEVELS: THE MECHANISM

GOLF XONE™ delivers high energy levels and alertness in humans, not by speeding you up, but by keeping you from slowing down.

Each time brain cells fire, they product a squirt of a chemical that serves as an off-switch that keeps neural activity in check.

GOLF XONE™ biochemically blocks the chemical, and jams the switch, so it can’t be turned down. This energy-effect can last from 2-4 hours.

Additionally, the time-released fruit glycoside system unique to the formula remains in the intestinal tract for a long period, providing increased energy over a 2-4 hour duration.

5-HOUR ENERGY CLAIMS: HIGH TECH IGNORANCE

Energy products claiming to deliver energy for more than a 2-4 hour period are absurd. The human brain and body function on a 4-hour time clock in terms of energy capacity, and no product can sustain more than 4-hours of energy. It is physically impossible.

5-hour energy claims are an example of High Tech Ignorance. GOLF XONE™ scientists refuse to make false and misleading claims and demand accurate scientific validation.

PHYSIOLOGICAL EFFECTS: FULL DISCLOSURE SCIENCE

GOLF XONE™ is effective at increasing energy, enacting thermogenesis via DIT, and adipose-tissue-fat-burning (ATFB), via the following mechanisms:

Stimulates under-functioning adrenal glands by increasing the output of epinephrine and norepinephrine from the adrenal glands.
Stimulates the release of catecholamines from the adrenal medulla and releases catecholamines due to a central action by and affecting C-AMP.
Donates methyl groups in anabolic processes (Rivici Index).
Increases energy expenditure dose dependently. Stepwise regression analysis with the thermogenic response as the dependent variable yields the following equation: (proprietary) as provided to our FDA-Regulatory attorneys for GOLF XONE™ Thermogenics validation of efficacy.
Turns on Brown-Fat-Thermogenesis in humans.
Removes adenos and phosphodiesterase blocks to thermogenesis in humans.
Triggers noradrenalin to active thermogenesis in isolated brown adipocytes by stimulating lipolysis.
Inclusion of Low Glycemic, thermogenic carbohydrates and PP glycosides that blunt Lipoprotein Lipase (LPL).
Inclusion of Buffered Caffeine (full U.S. Patent) that does not exhibit the adipose-tissue fat-storing properties of regular caffeine.

BODY OF RESEARCH
Human In Vivo Clinical Research & Trials

Anti-Carbohydrate properties, clinical studies, and metabolic effects in humans.
Low Glycemic Responses to specific Anti-Carbohydrate Fruit Sweeteners; Human In Vivo Clinical Trials.
Blood glucose effect of sugars, sweeteners and carbohydrates, circulating adiponectin (ACRP30), metabolic syndrome and sweeteners, genetic mutations in the leptin gene, adipocyte glucose metabolism, decreased glucose-induced thermogenesis (DGIT), circulating C peptide concentrations and insulin resistance, substrate utilization of carbohydrates and sweeteners.
The Insulin-Leptin-Ghrelin relationship in energy metabolism, improvement of glucose tolerance in type 2 diabetics in response to acute catalytic low-dose fruit glycosides, muscle glycogen and carbohydrates, Liporotein Lipase (LPL) and sugars.
Fruit Glycosides that do not trigger adipose tissue fat-storage, diabetes, or insulin resistance in humans, pathogenesis of obesity and Diet-Induced-Thermogenic (DIT) agents in humans.
Thermogenic and fat-storing response of carbohydrates, sugars and sweeteners, stimulation of fat-storing enzymes in humans, Brown Adipose Tissue (BAT) and thermogenesis, internal vs DIT thermogenesis, thermogenic capacity of cells and tissues.
UCP in mitochondria, Resistin, lipolytic actions in humans, appestat centers of the brain, identification and reduction of fat cell mass in humans, caloric conversion info fat cells, human genetic code related to deposition of adipose tissue body fat, N.E.A.T., fat thermostat in humans, hypothalamus-related fat-storage, cellular level thermogenesis, caffeine and DIT.
Methods for buffering caffeine to eliminate fat-storage in fat cells, caffeine thermogenesis, LPL gatekeepers for fat-storage in the fat cell, chromium and thermogenesis, chocolate and Serotonin-response in human female population.
Agents that activate Serotonin, high-protein diets and reduced thermogenesis, aging and adipose tissue fat accumulation, adipocyte lineage, regulation of beta-3-adrenoceptor expression in white vs brown fat cells.




PATENTED THERMOGENIC FAT-BURNING

The form of thermogenesis in humans activated by GOLF XONE™ is called Diet-Induced-Thermogenesis (DIT) and is well known in the scientific literature. GOLF XONE™ produces a DIT thermogenic effect, de novo lipogenesis, as well as carbohydrate oxidation (burning) in a Proprietary Low Glycemic matrix.

ANTI-GLUCOSE MATRIX

GOLF XONE™ contains a specially formulated ANTI-GLUCOSE MATRIX that addresses key areas in fat-burning metabolism:

Helps produce the neurotransmitter serotonin, which is related to positive moods.
Provides vital serum support in the Glucose-Producing-Reaction (GPR).


In order to combat Stress-Related-Eating (SRE) and False-Food-Cravings, the human body requires the opposite of Glucose-elevation, which is a Low Glycemic Matrix.

A Low Glycemic Matrix does not stimulate glucose, and conversely blocks False-Food-Cravings. The unique Matrix found in GOLF XONE™ provides a mechanism for blocking fat-storage and glucose-fat-storage.

The human body, and specifically the brain, requires at least 130 grams of carbohydrates per day for cognitive function and energy. Low energy levels and impaired cognitive function are typical when following a low carb diet.

High glycemic carbohydrates cause glucose and blood sugar swings, fat-storage, and weight gain.
High protein, and low carb diets and foods, exacerbate (worsen) the biochemical stress machine by depriving the body of stress-calming carbohydrates.

Thermogenic drinks and sports drink and compounds that do not contain a Low Glycemic Matrix cause fat-storage.


GOLF XONE™
PRODUCT USE GUIDELINES & CONTRAINDICATIONS

SUGGESTED USE:

Intended for adult use only. Not for use by individuals under 16 years of age. Not for use by children or by pregnant or lactating women. Use only as directed. Not for use by anyone sensitive to any of the ingredients.

This is a Sports-Specific beverage designed exclusively for Golfers and/or Class I Athletes (non-aerobic exercise, such as Golf, Race-Car-Drivers, Bodybuilders, Powerlifters). Not to be used for aerobic or intense sports or exercise.

AS A BEVERAGE FOR GOLFERS: Add one serving (one packet) to 16 ounces of plain bottled water (not flavored water or sports drinks) or to 16 ounces (oz) of plain water. Do not use more than 1 serving at a time. Do not use more than 4 servings total per day.

WEIGHT MANAGEMENT SUPPORT: GOLF XONE™ drink supports weight management in multiple ways: (1) Downregulation of Cortisol (2) Preferentially burns fat over carbohydrates (3) Downregulation of LPL (4) Non-Cephalic. Protocol for weight management support: Take 1 serving 30 minutes prior to a meal or in-between meals (up to 4 servings per day).

If dissolving into a full bottle of water, take a few sips before adding this product.

The energy-enhancement features of GOLF XONE™ may interfere with sleep if taken too close to bedtime. To avoid this, do not ingest after dinner or too close to sleep-hours.

PLEASE USE THIS PRODUCT RESPONSIBLY. Do not mix with alcohol or alcoholic drinks.

CAUTIONS: Do not exceed recommended serving size. Exceeding the recommended serving has not been shown to result in greater effectiveness. DO NOT USE IF YOU are pregnant or nursing. Keep out of reach of children. Do not use prior to intense aerobic sports or exercise.

CONSULT A PHYSICIAN BEFORE USING THIS PRODUCT IF YOU: Take a prescription medication, are sensitive to the effects of caffeine, or are allergic to any of the ingredients in this product.

Do not drink coffee or use caffeine products within 2 hours of taking this product. It is advisable to reduce consumption of products containing caffeine while taking this product. If you have a history of any of the following: heart disease, thyroid disease, diabetes, high blood pressure, glaucoma, difficulty in urinating or prostate enlargement, consult with your physician prior to using this product. DISCONTINUE USE AND CONSULT A PHYSICIAN IF YOU: Experience rapid heartbeat, dizziness, severe headache, shortness of breath or other similar symptoms.




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